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In this context, we also demonstrated that the disruption of the cellular redox buffer, controlled by GSH, increases not only the oxidative stress (Filomeni et al., 2005a), but also the endogenous physiological flux of NO in neuronal cells (Aquilano et al., 2011a, 2013)

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(1) F-actin formation is considered a key component in the cell's cytoskeleton, providing structure and aiding in cell movement