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In addition, administration of harvested endothelial progenitor cells from GPx1 −/− mice failed to augment revascularization in wildtype mice, whereas the wildtype endothelial cells improved blood flow and capillary density in the GPx1 −/− mice, suggesting that elevated levels of ROS in the GPx1-deficient cells are detrimental to endothelial progenitor cell function [77]