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Overall, increased oxygen consumption, energy production, and lactic acid production during high-intensity exercise can increase free radicals and oxidative stress, which can cause damage to cells and tissues if not adequately neutralized by the body’s antioxidant defense system

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Key comparative insights from the benzimidazole class: All benzimidazoles share CYP450-dependent hepatic metabolism, creating a common vulnerability pathway Hepatotoxicity risk across the class is dose-dependent and duration-dependent — consistent with the fenbendazole case reports Hepatotoxicity is generally reversible upon discontinuation across all benzimidazoles — also consistent with fenbendazole data The mechanism appears to involve both direct metabolite toxicity and immune-mediated components, particularly when combined with immunomodulatory agents Veterinary safety data for fenbendazole shows remarkable tolerability: studies in dogs have documented safety margins of 100× the standard dose without significant hepatotoxicity, suggesting that the human cases represent an uncommon susceptibility pattern rather than inherent high toxicity This class-level perspective reinforces that fenbendazole's hepatotoxic risk is real but predictable, dose-related, and manageable with appropriate monitoring — consistent with the safety profile of related compounds that have decades of human clinical use data