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FIGURE 6 Collectively, these data provide compelling evidence that GO@GSH-FA effectively mitigates the inflammatory response induced by OGD/R, as indicated by the marked reduction in the expression of key proinflammatory markers

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In this regard, excessive production of ROS, altered redox state [3] and a defect in regulation of apoptosis [4] are considered as imperative factors involved in the production, expansion of antibody flares and various clinical features in SLE